Alina’s death was a painful and preventable tragedy. Affectionately called “Ali” by her parents, she had a variety of recurring viral symptoms, starting with conjunctivitis at the age of 2 weeks, but was otherwise a bright and healthy girl. Repeated visits to various GPs always led to the same diagnosis: she had a virus that would clear up soon. Yet the cycle continued, and when she was 18 months old she suffered a seizure following her MMR vaccination and was admitted to Sydney Children’s Hospital (SCH).
This is a tragic story of how a Doctor can mistake a documented chronic viral illness with a genetic cause, how a young child can suffer unnecessarily from unsuitable medication and neglect, causing brain damage and eventually death. It shows how departmental colleagues will deliberately cause suffering to a child, and how a Hospital will assist in forcing a false diagnosis on parents to support a dishonest and incapable Doctor. The systems in place to protect patients and register complaints have no effect as they will only allow representation of events from the Hospital notes, which can easily shown to be false and incomplete
If you find yourself in a similar situation the following information will be of great help. The link between Chronic Enteroviruses (in this case EV71) , HFMD, Diabetes, Vaccine injury, glucose elevation levels and the Ketogenic diet, plus the use of appropriate Autoimmune drugs, like Anakinra. Also included in this diary is an explanation or examples of how the standard testing equipment used to detect viruses and autoimmune diseases fail
As parents we had battled with our daughters virus weekly, since her birth and over the course of 18 months to be met with a consistent reply from GPs “its just a virus, it will pass, return in 3 days if it hasn’t” (we often did, and we were told the same phrase again!)
One GP even advised us to be mindful of Viral complications, describing the red flags
However, for Alina the 40 visits to our GP and A&E were clearly the prodromal symptoms prior to her seizures. I have researched each, even the “discharge from eye” in her 1st week after birth, which developed into “Amblyopia, possible Estrophia” offers insight into how this virus develops.
I hope the slightly technical explanation below will help explain the associations, with developments and links to medical published papers within the diary of this webpage. The hope of this website is to raise awareness of medical malpractice, how it is hidden, and the explanation and treatment of a very obvious illness
A contradiction in care, a promoted adage by the British Medical Journals advice on Writing Medical Notes (2016) “if it isn’t written down, it didn’t happen.”
Unfortunately you will see that Dr Hugo Sampaio is overly reliant on this adage to hide his lack of knowledge, ability and procedure for a subject he is considered a Specialist. He will also come to lie to the medical board and HCCC while under investigation
As I gather, medical practice is largely based on an exclusion methodology, using tests to reinforce or reject an opinion or possibility, before trialling an alternative opinion or possibility.
I would imagine that one Doctor is reliant on another to have completed and documented their own assessment, as per the Medical Guide and expected practice.
I have noticed a generalised theme in the order of a medical diagnosis; Symptoms & clinical observations, History (to include extended family), initial opinion, testing to support this, preliminary diagnosis based on experience or consultation, then [suggested or accepted] remedy, revision or confirmation
Hospital Notes are the patients medical history, clinician observations, symptoms and responses. They are used for communication, continuation of treatment and quality of care, as well as decision making. A hospital is multi-discipline, with each Specialist type having access to review these notes. However this advantage is proved futile if the Lead Doctor deliberately withholds information. Furthermore a Specialist type would not find reason to question their testing equipment, or offer exceptions from their experience and knowledge.
Dr Hugo Sampaio was Alinas primary Doctor at Sydney Childrens Hospital, he did not take the first steps; accurate or documented clinical observations, or even medical history and symptoms. In fact he would often record the opposite, it also appears he was quite obstinate in jumping straight to opinion, then finding cause to dismiss any tests or opinions from colleagues that would contradict this.
He persisted beyond any reasonable expectation in particular of the opinion of this being caused by a mitochondrial gene error, such as POLG. I would consider this a sunken cost fallacy.
Dr Sampaio withheld information from the Hospital notes (noted especially with regards to the Ketogenic diet), from his colleagues and external consultants such as Dr Dale (a specialist in Immunology).
As parents this was one of our primary suspicions to his ability, we felt we were being gaslighted constantly as we were repeating the same agreed observations with him (and other Neurologists), with little or no action being taken, as a reasonable response, I started to record the conversations on my phone; to assure myself I had actually passed on the information, or had used understandable terms of expression
The numerous Audio and recordings and Videos are within the diary of this website, should you feel they are out of context please contact me for the full version. I have only clipped out unrelated conversations. They are otherwise in support of the information below
All tests within the Hospital failed to show any “signature” illness (other than an elevated Anti-GAD). Since I have only studied a couple of the tests in my former education, my interpretation of a correct diagnosis is based largely on Clinical Symptoms. This would seemingly be a disadvantage, but also I am not influenced by limitations of each testing equipment either.
Of the tests I have some knowledge of, finding fault in those is relatively simple, which I will develop on (there seems to be a significant historic fault in particular in the use of PCR from over 25 years ago). I understand it is the responsibility of the Specialist to know the limitations of their equipment, and from this I also conclude most tests of this type used by the Hospital are only able to identify acute variations of an illness, not the chronic. Alinas illness was most certainly chronic, evident in her GPs history (expanded within the diary), each of the symptoms are linked by one virus; Enterovirus EV71.
Enterovirus is a classification of viruses that originates from the gut (Entero – from within) they are very closely related to Rhino virus (a virus of the nose), so much that many tests cannot disgusting between them
During pregnancy Alinas mum was experiencing diabetic symptoms (which I discussed with Dr Sampaio – he replied pre-diabetes), significant and rapid deteriorating eyesight, drinking excessive amounts of water, hypothyroid (later re-evaluated as Hashimoto’s)
Previously mum had always avoided sugary foods as they would cause a rash. She also had what seemed to be an Acanthosis nigricans pigmentation on her neck, again mentioned to Dr Sampaio in an email
Alinas maternal grandfather is diabetic, and grandmother suffers from frequent pink eye, and multiple nerve palsys in both her left eye, and hips, she is frequently hospitalised for this.
On November 12th 2017, Alina was admitted overnight to a local hospital with heavy drooling and croup on her 1st seizure (limited to just left side of her throat and nowhere else, she was conscious and irritable). This was resolved immediately with IV midzolam.
Croup on her second seizure a couple of weeks later in her left arm and leg, and lip-smacking, this was again resolved immediately with IV midzolam. An MRI was taken and showed no brain inflammation. Again, sent home after
The third seizure, midzolam was administered intramuscular, it did not resolve the seizure. Also noted was a gait when she walked.
This all happened within a month prior to Alinas admission to SCH in late November 2017 under the care of Dr Hugo Sampaio. Brainstem inflammation was now recognised, and a focal seizure
Ataxia was also observed within the 1st week of ICU. The seizures remained, intermittent, or mildly continuous tonic-clonic movements in her left arm, she was otherwise alert, playful and active
In addition to this a summary of symptoms can be seen on the following paper
An email sent to Dr Sampaio in November between the 2nd seizure and admission to SCH, including images of a rash around mouth, Estrophic eye (dancing eye), and mentioning her jerking movements at night. This is as per the red flags described by Alinas GP, and very recognisable clinical symptoms caused by EV71. (a copy of the email and images are within the diary)
During initial assessment with both Dr Sampaio and Dr Sandi, as parents we still discussed Alinas medical history at length with the emphasis on the repeating virus & night time cough, wheeze, multiple visits to the GP and A&E (vomiting blood), visits to the ophthalmologist (under advice from an A&E Doctor) vaccine concerns & timing, croup, rashes. We had been to GPs too many times each to “simply forget” this repetition of illness. We were quite relieved to be able to discuss the full history with someone, following our frustrations with the GPs who were unwilling to escalate this
Following this Dr Sampaios only request was to measure all our head circumferences (done by Dr. Sandi)
With this long history of a repeating illness, he only noted that in the Hospital Notes that she was an “an otherwise well child, nil fevers/cough/cold symptoms” although even the transfer notes from the previous hospitals highlighted a long term cough and croup
In the Hospital notes he also writes;
Discussed with Prof Edwin Kirk. At this stage Alina’s presentation would not appear to be metabolic in nature … However, Dr Sampaio’s question of whether this could be POLG or mitochondrial cannot be completely excluded at this time
Very quickly during our stay, we noticed that sugar (glucose in the IV drip) would make the seizures worse, from here the ketogenic diet was implemented as a controlling measure in the second week of December. We explained at length how we noticed the first seizure not only coincided with her 18 month MMR, but also with the next stage of infant formula. Since Alinas mum had a poor reaction to sugar, this was limited in our household; Any time Alina had a high sugar item such as a macaroon or cake, she would develop rashes (like her mum). Baby formula was really the only source of “processed” Carbohydrates in her diet, everything else was made from the base ingredients
It was agreed by all the neurological team over the course of her stay that the ketogenic diet was highly effective in not only controlling the seizures, but also an indicator of her seizure activity; high seizure activity was associated with high glucose levels. Reduce seizure activity was associated with high keytone levels. Dr Sampaio even remarks himself. “Definitely! it’s the only valve”, yet not a single member of the Neurology team mentions this in the Hospital Notes
However it is all saved as a digital recording and can he heard within the diary section, the same observation commented on by ALL members of the Neurological department
It appears to me that Neurologists do not truly know why the ketogenic diet is effective. So I researched this too in relation to EV71 (it also holds for D68, with published papers suggesting it would be the same for other Enterovirus). An explanation of the relationship between these is within the diary pages
It is worth noting that a glucose IV drip is standard use in such admissions; and glucose proliferates this virus
After numerous conversations with Dr Sampaio regarding the persistent virus Alina had been suffering from, he said he had checked for the viruses we had mentioned. An email address for Dr Sampaio had been given to the parents on the 1st assessment prior to admission, and two emails of concerning features and images had been sent to him. The second of theses emails was an unmistakable description of EV71, including 2 facial images of the mouth and body rash caused by the virus
Mums concern was the timing of the 18 month MMR and the initial seizure, and a noticeable change in her personality and reluctance to eat, which Dr Sampaio dismissed as the EBV virus and the result was negative. The other virus (he did not name) he claimed to have requested a specialist test to check for this, and was met with the reply by myself “A Northern Blot?” to which he agreed. He also later said he would not trial Rituximab as it was a “horrible drug”
Dr Sampaio also said that he had given steroids during the second induced coma to rule out a viral cause anyway.
This of course left the parents confused, because we had been to the GPs and A&E too many times only to told “it was just a virus, it will pass” The same virus, which was getting worse each week over 18 months, then fading to only return again. One GP even gave a repeat prescription of 5x Steroids as our visits were so frequent
In the Hospital notes it is very clear that Dr Sampaio was still pursuing the belief this was POLG, he questioned that the genetic panel testing was insufficient (only looking for 3 known areas).
Sadly he did not question the limitations for the RT-PCR used by the Hospital (used to check for viruses)
The PCR array used at SCH is singled out in criticism for its lack of accuracy in identifying Enterovirus.
We naturally had no idea that Dr Sampaio had failed to contact Alinas GP, or vice-versa, since our GP had assured us she would (but an email to him included her contact information anyway). As parents we naturally expected Alinas medical history to be held on file, along with any direct email correspondence and of course the history given by the parents to include symptoms. (However parents cannot see the Hospital records as they are ‘confidential).
The policy of SCH regarding communications
”SCH states that it is not usual practice to obtain medical records from GPs, and often, the information required is available in the letters from the GP to the SCH. Oral history from the patient is obtained from the patient’s family upon initial assessment, and relevant information is documented in the medical records. The emails from family are considered and referred to the medical records where clinically relevant”.
The only test that showed any elevation was for a high Anti-GAD Immunological marker in the 2nd week of December, a CSF tap (Anti-GAD is associated with type 1, or autoimmune diabetes). This case strongly suggests that autoimmune diseases are not triggered by a chronic viral, but rather are the cause of an autoimmune disease (the testing equipment simply does not match or detect the nested version of a virus). A genetic cause was excluded by full genome testing
Dr Sampaio dismissed this anti-GAD marker as overlap or ‘bleed’ from IvIg given earlier in the week. We reminded him of the difficulties during pregnancy, and the maternal grandfather suffering from diabetes
Dr Sampaio consulted an external Immunologist; Dr Dale who was decisive in excluding an immunological explanation (and hence appropriate treatment). He too was excluded from knowledge of Alinas medical history. Dr Sampaio only suggests again this is a POLG-like illness
During a recorded conversation, in the judgement of whether Alina had an immunological condition, I was informed that Neopterin/Biopterins are the marker used, but they were considered within the normal range
However, there is a caveat to this opinion (as Dr. Dale discusses in his own published paper) it is effective-marker in identifying an immunologial issue except for a Chronic Progressive/Static range of biopterin levels. i.e. it is not applicable if the patient otherwise has a chronic illness (which Alina most certainly did!!)
By controlling the Ketogenic diet ratio (now at 4.0-4.5:1), and IV midzolam the seizures could be limited and the concerns for Alinas were downgraded. Alina was active, and as parents we were waiting desperately for her inflamed tonsils to be removed when the Surgeons returned from their Christmas vacation.
Dr Sampaio was told this illness was obviously originating from the throat by observations made from the parents, since it was the location of the 1st seizure (and the MRI taken at the time showed no cerebral inflammation) Alina also had a metallic smell on her breath prior to each episode of Croup, the smell was strongly present during a seizure
Over the Christmas period a conversation with the head of department Dr Bye, it was again explained the family relationship with diabetes and it was requested Alina be given Insulin. This was dismissed
Alina only received IVMP, and IVIG. As parents we realised the significance and family history and begged for Plasmapheresis and Rituximab, but this was declined as Dr Sampaio had committed himself to prove this was a genetic cause
Adenovirus was sweeping the ward after Christmas, on the 12th January 2018 the seizure went from isolated in the just the left arm to her full body within hours. Alinas limited seizure was now in a state of constant heavy seizure activity (likely cross immunity again). The focal seizure had become generalised.
Sadly Alina suffered very serious brain damage as a result, Dr Sampaio chose to go on holiday and ignore what was clearly a “very different seizure” (his own words). He was sent a video of the seizure and chose not to act. He waited 8 days before inducing a coma on this occasion as he felt he had over reacted with medications in the past, she suffered significantly and unnecessarily.
As parents we dismissed Dr Sampaio and requested care was taken over by a colleague Dr Cardomone; A ketogenic specialist, who himself later remarked “he couldn’t believe how responsive Alina was to the Ketogenic diet”
Dr Cardomone had abandoned the belief it was genetic. Dr Cardomone asked to revisit Alinas medical history, and based on the information he agreed to our request of the autoimmune drug Anakinra – as parents we had to submit a signed statement of symptoms and medical history, which we found odd; information we had already reported.
By this stage we had extreme fatigue and were struggling to recollect all points, especially as so many of our observations had been dismissed and “explained away” by testing/opinion/ or just simply ignoring them, gaslighting was very much used by the Neurological team. As parents we had been living in the hospital without family support for over 2 months, around the clock to ensure Alina was never alone.
A drug we had personally researched for this “unknown” disease and we believed to be the most successful based on clinical symptoms that we as parents had observed.
When we requested an Immunological/autoimmune treatment plan it was noted by an Immunologist team member
25/01 (Hospital Notes)
Question raised regarding possible immunodeficiency. Currently consider this unlikely as no significant personal or family history of infections.
Normal immunoglobulins and lymphocyte subsets
No evidence of cerebral inflammation on repeated CSF including low protein, no evidence of oligoclonal bands, normal neopterins in Nov 2017 and Jan 2018, CSF repeatedly negative for viruses on PCR. Repeat samples of CSF negative
for autoantibodies. No pleocytosis on CSF.
Note that evolving changes on MRI – encephalitis cannot be excluded, however, lack of CSF inflammation unusual.
NK cell function not clinically indicated as no evidence of CNS inflammation.
If viral PCR positive on brain biopsy, then further immune investigations / genetic testing may be warranted but not
It appears that Chronic EV does not appear in any of the above testing procedures used in an exclusion methodology
Since the Immunologist had no record of Alina significant personal and the family history of infections, there was no reason to question the validity of the testing equipment. For example
Outbreak of Neurologic Enterovirus Type 71 Disease: A Diagnostic Challenge – (2007 Carlos M Perez-Velez et al)
“…CSF viral culture results for patients with EV71-associated neurologic disease are reported to be positive for only 0%–3% of cases…”
www. americanbrainfoundation.org/neuroinflammation-and-brain-disease-meningitis-and-encephalitis
“…Encephalitis is an inflammation of the brain tissue. It can occur during or after an infection, including those transmitted by ticks and mosquitos. It can also develop if you have an autoimmune disease that causes your immune system to attack healthy brain tissue. This can sometimes be triggered by a vaccination or an infection, including the virus that causes COVID-19
Common viral causes of encephalitis include herpes simplex virus types 1 and 2, West Nile virus, tick-borne encephalitis, and enteroviruses, which can cause mild respiratory illnesses.
Encephalitis typically causes mild flu-like symptoms, though some people experience additional symptoms or no symptoms at all. These additional symptoms can include problems with speech, vision, or hearing, stiff neck, numbness or partial paralysis, and seizures.
As symptoms can worsen rapidly, doctors recommend seeking emergency medical attention for suspected cases of encephalitis.
While encephalitis can affect anyone at any age, it is more likely to occur in people with weakened immune systems, including infants, older adults, and people taking immune-suppressing medications…”
A trail was agreed of a low dose of Anakinra.
And it was incredibly successful, noted in the ICU records, but also clearly apparent in videos of comparison from before, and 10 days following the start of the trial. Alina could now track and trace, would withdraw her feet when blood samples were taken from her toes, significantly reduced cough and drooling as well as recognition of parents and even attempts to communicate. She was no longer in a vegetative state. The seizures were minimal during the day and completely absent when sleeping. A very stark contrast to before.
This caused significant issues for the Neurologists, in particular it would have brought to question and exposed Dr Sampaios lack of documentation, communication, ability and pursuit of a genetic cause for what was clearly an Autoimmune disease (that responded to autoimmune medication)
The Neurologists, now led by Dr Cardomone refused to extend the trial for what was a very clear improvement to her condition, untruthfully claiming she had worsened as a result of the trial.
Anakinra was withdrawn against the parents wishes. The Neurologists were forcing a return to the heavy and constant seizure state and obvious visual suffering, so we would request palliative care.
In February 2018 the full genome report was returned from America and it showed no gene concerns associated with EPC seizures. So the Hospital created one using a Insilica computer simulation, with focus on a very rare gene variation that both parents had, and was not the wild type. The software used is recognised by the developers themselves as being unreliable and over reporting this particular error. This gene error was extremely rare and happened to be in line with Dr Sampaio prediction, however there was very limited supporting information to this projection with only two publications available (and both with a significantly different presentation to Alinas condition).
So the Neurologists absolved themselves from blame by fabricating a genetic illness for which they could claim was fatal and beyond resolution, and validating Dr Sampaios diagnosis on admission.
2 years after Alinas death, a third paper was written on this “extremely rare gene” which proved it was not a valid diagnosis for Alinas condition in any way
In conclusion of the diagnosis, the balance of probability would confirm that Chronic EV71 is the same as Autoimmune Diabetes and can be treated successfully, like any virus it can be transmitted between family members, partners and also vertically during gestation. Diagnosis made by the methodology of exclusion (Bayes theorem and Occams razor), no positive test other than a highly elevated Anti-GAD and medical & family history. It was agreed by all Neurologists that the Ketogenic diet was the only effective treatment of Alina (but purposefully not documented). All other possibilities had been ruled out including a full Genome. The triggers were clearly the MMR, as well as elevated blood glucose and stress
Should there have been any doubt to the success of the procedural treatment the parents begged for (Chronic A71 related/Anti-GAD and highly responsive to the Ketogenic Diet) an almost duplicate case to Alinas condition was published in 2022, following a patient who’s presentation only differed in having Tonic-Clonic movements in his right arm, as opposed to Alinas left. Once you include the information from the transfer notes, the emails, images and points raised by the parents, the agreed effective medications (such as the Ketogenic diet), then the illness is very easily recognised. All this information is available within this web diary but not within the Hospital Notes
Favorable response to classic ketogenic diet in a child with anti-GAD 65 antibody mediated super refractory status epilepticus. (2022 Jun 7, Deepika Sivathanu et al)
“…A ketogenic diet has been sparingly used for the management of post-encephalitic epilepsy and autoimmune epilepsy. However, the data on the effectiveness of the ketogenic diet in the management of autoimmune encephalitis is scarce. Starting KD early in the disease course helped not only in seizure control but also preserved the cognitive and neurological well-being of the child.
A 7-year-old developmentally normal boy presented to the emergency room with a low-grade fever of 5 days duration followed by multiple episodes of seizures. The seizure semiology included lip-smacking movements and right upper limb tonic-clonic movements lasting for around 2 min.
Majority of children with autoimmune encephalitis present with seizures in the form of status epilepticus (SE) or super refractory status epilepticus (SRSE). SRSE is the SE that fails to terminate more than 24 h of anaesthetic infusion or recurs on weaning of the infusion. This child also presented with SRSE following a mild prodromal illness.
CSF oligoclonal bands, anti-TPO antibodies, ANA, and anti-NMDAR antibody titers were negative. While serum and CSF GAD-65Ab were positive (serum GAD-65 antibody titre: 221 IU/ml(0–17) and CSF GAD-65 AB titre:218 IU/ml [0–17]). Hence a diagnosis of anti-GAD-65 Ab-associated AIE was made.
On day 7, second-line immunomodulation therapy with rituximab (750 mg/m2) was given
The child is on regular follow-up for more than a year. A serial clinical examination with developmental assessment is being performed at every visit.
He remains seizure-free and can attend regular school with good scholastic performance. His Modified Rankin score was 1 at the last follow-up…”
To note, this was written regarding a case 4 years prior, in either 2017-2018; Dr Sampaio had the same exposure to current research as the author Dr Deepika
Incidentally, Alina had spent a total of 104 days in ICU. The parents were told the cost was an approximate $5000/day. The overall cost to include an overseas genome report and multiple visits to several other departments and medications likely doubled this
Without Alinas Medical history or correct clinical observations, none of the multidisciplinary reviews by colleague Doctors would be able to even make a close diagnosis or offer a suggestion from their experience.
In a statement to the Medical Board, Health Care Complaints Commission (and Child Death Review Team) Dr Sampaio claimed that Alina showed no apparent benefit to Anakinra, however the videos and ICU notes show otherwise. It was also evident by comparison between the EEGs
He stated that Alina was refractory to the ketogenic diet, but in contradiction, he himself was using it as a guide to her seizure status, impressed by its benefits in controlling the seizures, and going to great lengths to protect its application.
He lied to them
The Hospital Notes had clearly been written with a view to avoiding any future liability for her treatment. None of the investigation teams had reason to doubt him, as he had simply excluded this information from the Hospital notes.
“if it isn’t written down, it didn’t happen.”
The HCCC and Medical Board were offered the recordings, but refused their use (no reason was given). A statement was written, but I was verbally told that it would not be considered “as my memory would have been affected by such a stressful situation”
Dr Sampaio was praised for his handling of such a complex case
Further attempts at bringing the hospital staff to justice were made by approaching lawyers, the Coroner, APHRA, the Minister of Health and even the Police. All turned up blanks and Alina’s father was told that there was no proof of any mistreatment, as any legal case would rely solely on the hospital notes and the word of the Neurological team, and the audio and video recordings which clearly showed a genuine history of events would not be admissible as evidence.
After the third communication to the Minister of Health, I was referred to the Ombudsman. I presented them with his understanding of the law, that in NSW Australia you are allowed to make recordings if you believed you are protecting your own interests, further defined by the Supreme Courts in 2015 as a “genuine fear for your safety” (Groom v Police [2015] SASC 101)
The Ombudsman said their “legal team” did not agree with this interpretation, but didn’t elaborate with any reasoning, explanation or examples
In Australia, investigation of Criminal activity is reliant on the opinion of the either the Police or Medical Professionals. Both of these are afforded the authorisation and discretion to select evidence that they consider relevant and disregard anything to the contrary, such as audio and video to demonstrate a Doctor had failed to uphold expected medical practice, and positioned himself in the medical notes to submit false statements to the investigation teams.
Intermediate groups such as APHRA, the Coroner, HCCC or Child Death Review Team will not overstep the opinion of another regulatory body
Members of Parliament, such as the Minister of Health, will distance themselves from intervention by simply referring the matter to an organisation such as the Ombudsman, who have neither the power to intervene, nor offer Legal advice.
You can imagine the cyclic nature of being passed from one department to another and the painstaking long time between contact and response, if one is made at all!
In Australia vaccinations are compulsory. Australia appears to have agreed to the Humans Rights declaration for “right to life” like many other nations, however there is no method for actually raising a concern or complaint
For the last 5 years, Alina’s father has continued researching and painstakingly examining every aspect of her treatment in preparing this website. He remains more convinced than ever that the stubborn, negligent and continual misdiagnosis of her illness as genetic rather than viral, ultimately led to her death. By the end of her life she was so far brain damaged by the erroneous treatment that she would have likely required a lifetime of 24/7 care, something the hospital would never want to be held responsible for. So the Hospital withdrew effective care and fabricated a genetic cause to disguise and confuse the horrendous error of the Lead Doctor, a Doctor who is now currently the Departmental Head of Paediatric Neurology.